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As summarized earlier, ferroptosis arises from a dysregulated balance between pro-ferroptotic cues (e.g., labile iron accumulation and PUFA enrichment) and protective mechanisms, including the SLC7A11GSHGPX4 axis, GCH1BH4 pathway, FSP1CoQH2 system, DHODHCoQH2 cascade, and MBOAT1/2-mediated monounsaturated fatty acid (MUFA) synthesis (280282)

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