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timonensis led to a transient accumulation of BB that was subsequently consumed with concomitant production of TMA (Figure 9A, top)

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Mutations in these genes are linked to the regulation of amyloid-beta processing, a crucial component of AD pathogenesis

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showed that pregnant and prepregnant females with G6PD deficiency have higher blood pressure values, especially that of systolic blood pressure, compared with females with a normal enzymatic activity [16]