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While early anecdotal evidence suggested significant clinical symptomatic improvements, the five clinical trials identified in a recent meta-analysis used small populations with different clinical syndromes, measured different outcomes, and yielded no overall evidence of benefit [109]

AA is a basic precursor of numerous lipid-based signaling molecules and can be further transformed by cyclooxygenases (COX), lipoxygenases (LOX) and cytochrome p450 (CYP450) to form eicosanoids, further influencing redox balance and inflammation by engaging specific receptors 93

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Lean, metabolically healthy profiles use roughly 0.3 to 0.5 mg because exposure and heart-rate sensitivity are higher at the same nominal dose. These sub-1 mg ranges are modeled rather than outcome-tested

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