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World J Gastroenterol 14(26):41014110

These findings illustrate the therapeutic potential of dual-pathway intervention, although the complex interplay between autophagy, ferroptosis and various immune cell populations requires further mechanistic elucidation to optimize combination strategies

Hence, although future kinetic studies might confirm a random mechanism for the rate-limiting first part of monothiol mechanism (iii) in vitro, GSH is probably the first and the non-glutathione disulfide the second substrate under physiological conditions in vivo (Fig

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6f) in HepG2 cells