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In the PWA model, GE suppressed hypothalamic expression of BCAP31, a pro-apoptotic protein, and promoted proteins associated with autophagy-critical processes for neuronal function and energy homeostasis

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Notably, like other cholesterol biosynthesis inhibitors, triparanol activated transcription of cholesterol biosynthetic genes SQLE and FDFT1 , indicating a functional consequence of DHCR24 inhibition (Supplementary Fig