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Fraternale A, Paoletti MF, Casabianca A, et al

Importantly, Epitalon treatment significantly reduced H2AX expression, suggesting that the peptide alleviates aging-induced genomic stress [2]

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In turn, the over-expression of p-160 co-activators has been shown to enrich E2-mediated gene expression and improve cell survival by suppressing the tumor necrosis factor (TNF) activity (Yu et al., 2012), supporting the participation of estrogens in the maintenance of cell survival