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Distinct Cysteine Residues in Keap1 Are Required for Keap1-dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress

Inflammation, increased oxidative and metabolic stress, endothelial dysfunction, vascular calcification resulting from poor calcium and phosphate metabolism, and difficulties with coagulation contribute significantly to the pathogenesis of CKD- and ESKD-related complications

Jim Stoppani, Ph.D

Percentages of proliferative (BrdU + ) (n = 6 technical replicates representative of 2 independent experiments) ( a ) and apoptotic (Annexin V + ) (n = 3 technical replicates representative of 3 independent experiments, **p = 0.0042) ( b ) cells were determined

10.1038/s41467-018-05655-x Nat