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Mechanistic analyses revealed that cotadutides enhanced hepatic benefits were associated with increased mitochondrial -oxidation capacity, improved mitochondrial respiratory chain function and more pronounced suppression of de novo lipogenesis enzymes

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Iron accumulation and lipid peroxidation in -cells, driven by mitochondrial dysfunction and inflammatory cytokines like IL-1 and IFN- produced by T cells and macrophages, lead to -cell dysfunction and apoptosis (105, 106, 109, 146)

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Front Endocrinol (Lausanne) 2021;12:649018