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F., Fulton, S

Concurrently, early microangiopathy and increased endothelial permeability have been identified, even in initial disease stages, marked by disrupted tight junctions and altered endothelial behavior

The mechanistic interpretation, debated for two decades, was that GIPR agonism did two things: it contributed to appetite suppression and energy expenditure in addition to GLP-1R activation, and - separately - it appeared to improve tolerability by reducing GLP-1-mediated nausea and GI adverse events