Since the mevalonate pathway is closely related to ferroptosis through regulating expression of GPX4, production of squalene and coenzyme Q, and sterols are known to exert intricate feed-back regulations on this pathway to restrict cholesterol overproduction, we hypothesized that cholesterol and its metabolites would modulate cell sensitivity to ferroptosis
The result is a peptide that retains structural integrity in environments, gastric fluid, intestinal lumen, serum, where conventional peptides are inactivated within minutes
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