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Its senescence-related actions are downstream of the telomere checkpoint reducing SASP and cellular dysfunction that telomere shortening triggers rather than addressing the shortening itself

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For example, in a thioacetamide-induced mouse model, semaglutide reduced biochemical and histological markers of liver injury, attenuated -SMA expression, decreased oxidative stress, and downregulated the canonical TGF-/Smad signalling pathway while upregulating SIRT1 and phosphorylated AMPK