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These results therefore clarified blood oxidative stress profile in children with ASD, strengthening clinical evidence of increased oxidative stress implicating in pathogenesis of ASD

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12 Moreover, lipotoxicity, endotoxin translocation, and chronic low-grade inflammation activate Kupffer cells (KCs) and polarize immune cell populations, 13 contributing to a persistent pro-inflammatory hepatic microenvironment

6.6.7.4 Ghrelin In chronic DSS-induced colitis in ghrelin KO mice, due to the absence of endogenous ghrelin, the DAI was reduced, and the infiltration of neutrophils was delayed compared to wild-type (300)