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*Correspondence: Xin Yan, [email protected] These authors have contributed equally to this work This article was submitted to Obstetric and Pediatric Pharmacology, a section of the journal Frontiers in Pharmacology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Previously, there is study revealed that promoting cellular intake of riboflavin by upregulating expression of SLC52A1 (a riboflavin transporter, also known as GPR172B/RFVT1) plays a crucial role in antisenescence via activating mitochondrial membrane potential and inhibiting the AMPK-p53 pathway [56]

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Experiments were performed in quadruplicates using cultures derived from three different human fetal brains