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It strongly binds remaining toxins and plays a role in aiding antidotes such as N-acetylcysteine (NAC) for paracetamol toxicity
doi: 10.1007/s00011-010-0245-x 33 BrownDIGriendlingKK
Since cholesterol levels in cells are tightly controlled (cholesterol is critical to many cellular functions), the shutdown of cellular cholesterol synthesis causes the cell to respond by increasing the activity of the LDL receptor on the cell surface, which has the net effect of pulling LDL particles out of the bloodstream and into the cell
It does not simply regenerate lost tissue in a straightforward way
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