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10.1159/000322399 159 WangZ.WangY.WangZ.GutkindJ

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As the disease advances, ferroptosis-induced release of damage-associated molecular patterns (DAMPs), exosomal transmission, and immune evasion mechanisms further amplify neuroinflammation and cell damage, establishing a cascade of intercellular pathological propagation that accelerates neurodegeneration

It regulates the expression of tight junction proteins, which close the gaps between intestinal cells to prevent the leakage of toxic substances into the bloodstream and subsequent systemic inflammation