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[6] Research shows that these effects are dependent on PepT1, a peptide transporter that becomes upregulated in inflamed intestinal tissue

Therefore, while aware that the pharmacological modulation of the NRF2/ARE pathway could undeniably offer new therapeutic opportunities for diseases related to OS, we should at the same time consider that most of the currently known activators of NRF2 are not particularly specific, leading to an increased risk of off target toxic effects

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Research using the Broad Institute's Connectivity Map found that GHK-Cu influences the expression of over 4,000 human genes - including many involved in repair, anti-inflammatory response and extracellular matrix maintenance