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Bloodbrain barrier disruption in PD has been modeled with a human BBB-on-chip cultivated under flow conditions and containing iPSC-derived dopaminergic neurons, astrocytes, microglia, and pericytes differentiated with cell-specific chemically defined medium and hBECs using Qian's method from patients

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l-carnitine induced AMPK phosphorylation and expression to protect against synovitis and delayed OA progression in the ACLT-induced model The results of the dissociative experiments prompted us to further investigate the anti-inflammatory potential of l-carnitine in animal experiments

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