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The taxonomical features (i.e., phyla and genera) were analyzed via carrying out a linear mixed model [46] on all the features in the data set, whilst taking into account the false discovery rate (type I error) under repeated testing through Benjamini & Hochberg correction [49], of the P-values calculated via type III ANOVA using the Satterthwaites method [47]

In the case of epilepsy, impaired K + buffering, glutamate clearance, lipid accumulation and malfunctioning of the glutamate (GABA)-glutamine shuttle emerge as leading pathophysiological processes