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193 In addition, Fusobacterium nucleatum can activate the RIPK1-mediated apoptosis pathway in UC, resulting in the destruction of tight junctions between cells, which in turn promotes the destruction of the intestinal barrier, inhibits the renewal of intestinal epithelial cells, and accelerates the progression of UC

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Gel-shift analysis revealed reduced LXR binding activity in the I148M oe M1 macrophage, leading to the downregulation of ABCA1 (-36.7%) and ABCG1 (-32%)

Clinical trials demonstrate that maximal therapeutic effects generally require several months of treatment at the target maintenance dose, which is typically reached through a gradual dose escalation process

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