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However, in contrast to patients with chronic GH excess (acromegaly), where sKL is greatly elevated, in treated AGHD patients it remains within the normal range also seen in normal subjects and NFPA patients

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And in the brain, it crosses the blood-brain barrier and activates anorexigenic neurons in the hypothalamus, specifically the POMC/CART pathways that signal fullness while suppressing the NPY/AgRP pathways that drive hunger