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Hepatocytes can adapt their enzymatic machinery in response to exposure to toxicants, a process called induction that enhances protection towards elevated levels of circulating harmful compounds [15]

Primary hepatocytes from mice lacking cysteine dioxygenase show increased cysteine concentrations and higher rates of metabolism of cysteine to hydrogen sulfide and thiosulfate

This leads to severe oxidative stress, lipid peroxidation, and catastrophic damage to cellular membranes and mitochondrial DNA, triggering systemic inflammation