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To further establish mitochondrial impairment as a key driver of skewed epithelial differentiation processes, we used a mouse model with an epithelial-specific knockout of caseinolytic mitochondrial matrix peptidase (ClpP IEC )

Myocardial remodelling characterized by fibrosis and hypertrophy, impaired microcirculation, abnormal protein function caused by advanced glycation end products, changes in myocardial metabolism and abnormal mitochondrial function have been noted as associated factors of diabetic cardiomyopathy [14]

2018;1864(9 Pt B):2814-2821

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Glucagon-like peptide 1 receptor agonists: a role in Glaucoma