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The antibiotic nitrofurantoin (NFT) was identified to stimulate de novo synthesis of glutathione in the human hepatoma cell line, HepG2, and in primary human hepatocytes
In turn, these pathological changes lead to airflow limitation and the other physiological abnormalities characteristic of COPD
Acetaminophen readily crosses the blood-brain barrier, and the central nervous system (CNS) is considered to be the primary site of action of the drug [1]
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