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Tendon injuries initially suffer from: Poor blood supply Limited vascularity Slow nutrient delivery In rodent models, BPC-157 appears to: Upregulate VEGFR2 signalling Increase capillary density Improve microvascular perfusion Mechanistically: BPC-157 VEGFR2 activation PI3K/Akt pathway eNOS phosphorylation Increased nitric oxide Improved angiogenesis More vascularity = better nutrient and oxygen delivery to fibroblasts
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Figure 3 Omega-3 fatty acids Among the nutritional intakes, fatty acids (FAs) have a crucial role in determining the composition of lipid-content cell membranes and even cell signaling molecules (124, 125)
investigated in models of traumatic brain injury, Parkinsons-related pathways, and neurological inflammation Nitric oxide system interaction Proposed to modulate NO synthesis pathways
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