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The researchers proposed that the compounds effects on muscle function operate through boosting NAD+ levels by inhibiting NNMTs consumption of nicotinamide, more substrate is available for NAD+ synthesis via the salvage pathway, supporting mitochondrial function and energy metabolism in aged skeletal muscle

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For manual annotation, we obtained markers using the FindClusters function from Seurat (Supplementary Data File 1 Top 50 cell markers in each cluster ) and we observed cell-type-specific expression of a number of genes (Fig

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Matrix metalloproteinase-9 delays wound healing in a murine wound model